Acquired Horner Syndrome is a medical condition characterized by a collection of symptoms resulting from damage or disruption to the sympathetic nerve pathway in the body. This condition leads to the partial or complete dysfunction of the sympathetic nervous system, which is responsible for regulating various bodily functions.
Horner Syndrome itself refers to a set of clinical signs and symptoms that include drooping of the upper eyelid (ptosis), constriction of the pupil (miosis), sinking of the eyeball into the eye socket (enophthalmos), and decreased sweating on the affected side of the face (anhidrosis). These symptoms typically occur on one side of the face and can be caused by various underlying conditions or injuries.
When Horner Syndrome is acquired, it implies that it is not present at birth but develops later in life. The acquired form of this syndrome can arise due to various causes, such as trauma, tumors, surgery, vascular disorders, or neurological damage. The specific cause of acquired Horner Syndrome may differ depending on the individual case.
Diagnosis of acquired Horner Syndrome involves a comprehensive evaluation of the patient's medical history, a thorough physical examination, and additional tests, such as imaging studies or nerve conduction studies, to identify the underlying cause. Treatment focuses on addressing the root cause of the condition and managing the symptoms accordingly. This may involve surgical interventions, medications, or other appropriate therapies depending on the specific cause and severity of the syndrome.
Overall, acquired Horner Syndrome is a condition characterized by a set of specific ocular and facial manifestations resulting from damage or dysfunction of the sympathetic nervous system, with various potential causes and treatment approaches.
