Rheumatic gout, also known as pseudogout or calcium pyrophosphate dihydrate (CPPD) crystal deposition disease, is a type of arthritis characterized by sudden and painful joint inflammation. It is caused by the accumulation of calcium pyrophosphate crystals in the joints and surrounding tissues.
The term "rheumatic gout" is often used to describe this condition because the symptoms resemble those of classic gout, including severe pain, swelling, redness, and warmth in the affected joints. However, rheumatic gout differs from typical gout in terms of its underlying cause and the crystals involved. In gout, uric acid crystals accumulate in the joints, while in rheumatic gout, calcium pyrophosphate crystals are the main culprit.
Rheumatic gout primarily affects the knees, wrists, ankles, and other weight-bearing joints. The attacks are usually acute and may last for several days to weeks. Individuals with rheumatic gout may also experience stiffness, limited range of motion, and joint deformities over time.
The exact cause of rheumatic gout is not fully understood, but several factors increase the risk, including age, trauma, other joint conditions (such as osteoarthritis), and certain metabolic disorders. Diagnosis typically involves a combination of medical history, physical examination, joint fluid analysis, and imaging tests.
Treatment for rheumatic gout aims to relieve pain, reduce inflammation, and prevent further crystal accumulation. This may involve nonsteroidal anti-inflammatory drugs (NSAIDs), corticosteroid injections, colchicine, and lifestyle modifications. In some cases, joint aspiration or surgery may be necessary to remove the accumulated crystals.
Managing rheumatic gout usually focuses on long-term strategies to minimize symptoms and prevent future flares. Regular exercise, maintaining
