Regular gout, also known as primary gout or idiopathic gout, is a form of arthritis characterized by sudden and recurrent episodes of intense joint pain, swelling, redness, and tenderness. It is caused by the accumulation of uric acid crystals in the joints and surrounding tissues, leading to inflammation and discomfort. This condition primarily affects the feet, particularly the big toe, but can also occur in other joints such as the ankles, knees, elbows, wrists, and fingers.
Regular gout typically occurs in individuals with high levels of uric acid in their blood, a condition known as hyperuricemia. Uric acid is a waste product produced by the breakdown of purines, which are substances found in certain foods and beverages. When the body produces excessive uric acid or fails to excrete it efficiently, the excess uric acid accumulates and forms crystals in the joints.
The onset of regular gout attacks is often sudden and intense, often during the night. The affected joint becomes extremely painful, swollen, and tender to the touch. The pain usually peaks within several hours and can last for a few days to several weeks. In between the attacks, individuals with regular gout may be symptom-free for weeks, months, or even years.
Treatment for regular gout involves managing acute attacks through the use of nonsteroidal anti-inflammatory drugs (NSAIDs), corticosteroids, or colchicine to reduce pain and inflammation. Lifestyle modifications such as maintaining a healthy weight, following a low-purine diet, limiting alcohol consumption, and staying well-hydrated can help prevent future gout attacks. Additionally, medications like urate-lowering drugs may be prescribed to manage high uric acid levels and prevent the recurrence of regular gout episodes. Regular monitoring
